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Vitamins and minerals

Calcium tablets — do they end up in the bones or the arteries?

Sources: 5 · Article: 4 495 characters

Jerzy Zięba points out that the cited studies linked calcium supplements to a higher risk of arterial calcification, heart attack and stroke.

  • Osteocalcin incorporates calcium into the bones.
  • Jan Jaśkowski advises against taking extra calcium supplements to avoid calcification of soft tissues, including blood vessels.
  • Among other things, vitamin D3 is responsible for the proper absorption of calcium from the blood and, in turn, for building bones.

What the authors write

Jerzy Zięba

The studies cited by Jerzy Zięba linked calcium supplements to a higher risk of arterial calcification, heart attack and stroke.

„Many readers may find it strange that recently published studies (2011) indicate that women who took calcium supplements to prevent osteoporosis were more likely to develop calcification in their arteries, have a heart attack or suffer a stroke than women who did not take calcium supplements.”Dla wielu czytelników dziwnym wyda się fakt, że niedawno opublikowane badania (2011) wskazują, że kobiety, które brały suplementy wapnia, po to, żeby zapobiec osteoporozie były bardziej narażone na wytworzenie zwapnień w tętnicach, zawał serca czy udar mózgu, niż kobiety, które suplementów wapnia nie brały.
Jerzy Zięba

Osteocalcin incorporates calcium into the bones.

„Special cells called osteoblasts release a substance called osteocalcin, which incorporates calcium into the bones. Other cells, called osteoclasts, remove old, worn-out cells. If osteoclast activity becomes dominant, more cells are removed than are incorporated.”Specjalne komórki tzw. osteoblasty, wydzielają substancję zwaną osteokalcyną, która wbudowuje wapń do kości. Inne komórki tzw. osteoklasty usuwają komórki stare, zużyte. Jeśli nastąpi przewaga działania osteoklastów, wtedy więcej komórek ulega usunięciu niż wbudowaniu.
Jan Jaśkowski

Jan Jaśkowski advises against taking extra calcium supplements to avoid calcification of soft tissues, including blood vessels.

„Remember to take vitamin D supplements with vitamin K2 to avoid calcification of soft tissues, including blood vessels. For this reason, you should NOT take additional calcium supplements.”Należy pamiętać aby witaminę D suplementować z witaminą K2, aby uniknąć zwapnienia tkanek miękkich w tym naczyń krwionośnych. Z tego też powodu NIE należy suplementować dodatkowo wapnia.
Jan Jaśkowski

Among other things, vitamin D3 is responsible for the proper absorption of calcium from the blood and, in turn, for building bones.

„Among other things, vitamin D3 is responsible for the proper absorption of calcium from the blood and, in turn, for building bones. It is widely known that older people are at risk of osteoporosis and more frequent bone fractures. Unfortunately, in older people, as many as 21% of hip fractures result in the patient's death within a year of the fracture!”Witamina D3 jest między innymi odpowiedzialna za prawidłowe wchłanianie wapnia z krwi, a tym samym za budowę kości. Powszechnie wiadomo, że osoby starsze są narażone na osteoporozę i częstsze złamania kości. Niestety u osób starszych nawet 21% złamań kości udowej kończy się śmiercią pacjenta w ciągu roku od złamania!

Perspectives and opinion materials

The content below presents the authors’ opinions, interpretations and hypotheses. It is not treated as equal to scientific studies and systematic reviews.

Orthomolecular Medicine News Service Opinion material
„The reason people die of cardiovascular disease usually begins with inflammation and progressive calcification, not cholesterol levels. The correlations between inflammation, calcification and death by cardiovascular disease (CVD) are much stronger than the correlations between cholesterol levels and death by CVD (Bolland et al., 2008). Following the guidelines of the American Heart Association (AHA) may be useful for liability and good business, but is not always useful for maintaining optimal health. To understand what is happening inside the body, just look at images of vascular calcification.”The reason people die of cardiovascular disease usually begins with inflammation and progressive calcification, not cholesterol levels. The correlations between inflammation, calcification and death by cardiovascular disease (CVD) are much stronger than for correlations between cholesterol levels and death by CVD (Bolland et al, 2008). Following the guidelines of the American Heart Association (AHA) may be useful for liability and good business, but is not always useful for maintaining optimal health. To understand what's going on inside of the body, just take a look at the images of vascular calcification.
Orthomolecular Medicine News Service Opinion material
„(2010) Estrogen inhibits vascular calcification via the vascular RANKL system: common mechanism of osteoporosis and vascular calcification. Circ Res. 107:466-475. Das UN (2002) Nitric oxide as the mediator of the antiosteoporotic actions of estrogen, statins, and essential fatty acids. Exp Biol Med.”(2010) Estrogen inhibits vascular calcification via vascular RANKL system: common mechanism of osteoporosis and vascular calcification. Circ Res. 107:466-475. Das UN (2002) Nitric oxide as the mediator of the antiosteoporotic actions of estrogen, statins, and essential fatty acids. Exp Biol Med.
Orthomolecular Medicine News Service Opinion material
„Additional clinical observations, including the long-term maintenance of excellent bone density and low coronary calcium burden in the author and several other patients, further support the hypothesis that bone health reflects broader metabolic, nutritional, hormonal and vascular physiology rather than calcium metabolism alone. Representative clinical observations from our practice are summarized in Table 1. These cases demonstrate improvements in bone density occurring alongside improvements in vascular health markers, including carotid intima-media thickness (IMT) and coronary artery calcium (CAC) scores.”Additional clinical observations, including long-term maintenance of excellent bone density and low coronary calcium burden in the author and several other patients, further support the hypothesis that bone health reflects broader metabolic, nutritional, hormonal, and vascular physiology rather than calcium metabolism alone. Representative clinical observations from our practice are summarized in Table 1. These cases demonstrate improvement in bone density occurring concurrently with improvement in vascular health markers, including carotid intima-media thickness (IMT) and coronary artery calcium (CAC) scores.

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